GPR154 antibody (clone 2F5), anti-human
€435.00
In stock
SKU
AC-AT2245a
Catalog Number: AC-AT2245a
Size: 100 µg
Isotype: mouse IgG1 Kappa
Applications: WB, E
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Size: 100 µg
Isotype: mouse IgG1 Kappa
Applications: WB, E
Request Information AC-AT2245a">Request Information
Background:
This gene is a member of the G protein-coupled receptor 1 family and encodes a plasma membrane protein. Increased expression of this gene in ciliated cells of the respiratory epithelium and in bronchial smooth muscle cells is associated with asthma. Mutations in this gene have also been associated with this disease. Alternatively spliced variants which encode different protein isoforms have been described; however, not all variants have been fully characterized.
Other Names:
Neuropeptide S receptor, G-protein coupled receptor 154, G-protein coupled receptor PGR14, G-protein coupled receptor for asthma susceptibility, NPSR1, GPR154, GPRA, PGR14
Antigen Type:
Recombinant Protein
Gene Name: NPSR1
Gene ID: 387129
NCBI Accession: NP_997055.1;NP_997056.1
Primary Accession: Q6W5P4
Other Accession: NM_207173
Format: Clear, colorless solution in phosphate buffered saline, pH 7.2 .
This gene is a member of the G protein-coupled receptor 1 family and encodes a plasma membrane protein. Increased expression of this gene in ciliated cells of the respiratory epithelium and in bronchial smooth muscle cells is associated with asthma. Mutations in this gene have also been associated with this disease. Alternatively spliced variants which encode different protein isoforms have been described; however, not all variants have been fully characterized.
Other Names:
Neuropeptide S receptor, G-protein coupled receptor 154, G-protein coupled receptor PGR14, G-protein coupled receptor for asthma susceptibility, NPSR1, GPR154, GPRA, PGR14
Antigen Type:
Recombinant Protein
Gene Name: NPSR1
Gene ID: 387129
NCBI Accession: NP_997055.1;NP_997056.1
Primary Accession: Q6W5P4
Other Accession: NM_207173
Format: Clear, colorless solution in phosphate buffered saline, pH 7.2 .
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