Kir3.3 polyclonal, anti-human, mouse, rat
€470.00
In stock
SKU
AC-AP51300
Catalog Number: AC-AP54246
Size: 100 µl
Host: Rabbit
Applications: WB, IHC-P, IHC-F, IF, E
Datasheet
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Size: 100 µl
Host: Rabbit
Applications: WB, IHC-P, IHC-F, IF, E
Datasheet
Request Information
Background:
This receptor is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium (By similarity).
Other Names:
G protein-activated inward rectifier potassium channel 3, GIRK-3, Inward rectifier K(+) channel Kir33, Potassium channel, inwardly rectifying subfamily J member 9, KCNJ9, GIRK3
Antigen Type: synthetic peptide
Antigen Type: KCNJ9
Gene ID: 3765
Primary Accession: Q92806
This receptor is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium (By similarity).
Other Names:
G protein-activated inward rectifier potassium channel 3, GIRK-3, Inward rectifier K(+) channel Kir33, Potassium channel, inwardly rectifying subfamily J member 9, KCNJ9, GIRK3
Antigen Type: synthetic peptide
Antigen Type: KCNJ9
Gene ID: 3765
Primary Accession: Q92806
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