Kir3.4 polyclonal, anti-human, mouse, rat
€470.00
In stock
SKU
AC-AP51951
Catalog Number: AC-AP54896
Size: 100 µl
Host: Rabbit
Applications: IHC-P, IHC-F, IF, ICC, E
Datasheet
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Size: 100 µl
Host: Rabbit
Applications: IHC-P, IHC-F, IF, ICC, E
Datasheet
Request Information
Background:
This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium.
Other Names:
G protein-activated inward rectifier potassium channel 4, GIRK-4, Cardiac inward rectifier, CIR, Heart KATP channel, Inward rectifier K(+) channel Kir34, IRK-4, KATP-1, Potassium channel, inwardly rectifying subfamily J member 5, KCNJ5, GIRK4
Antigen Type: synthetic peptide
Antigen Type: KCNJ5
Gene ID: 3762
Primary Accession: P48544
This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium.
Other Names:
G protein-activated inward rectifier potassium channel 4, GIRK-4, Cardiac inward rectifier, CIR, Heart KATP channel, Inward rectifier K(+) channel Kir34, IRK-4, KATP-1, Potassium channel, inwardly rectifying subfamily J member 5, KCNJ5, GIRK4
Antigen Type: synthetic peptide
Antigen Type: KCNJ5
Gene ID: 3762
Primary Accession: P48544
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