Phospho-TAL1(T90) polyclonal, anti-human
€0.00
In stock
SKU
AC-AP3690a
Catalog Number: AC-AP3690a
Size: 80 µl, 400 µl
Isotype: Rabbit IgG
Clone Name: RB15311
Applications: DB
Datasheet
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Size: 80 µl, 400 µl
Isotype: Rabbit IgG
Clone Name: RB15311
Applications: DB
Datasheet
Request Information
Background:
TAL1 is implicated in the genesis of hemopoietic malignancies. It may play an important role in hemopoietic differentiation and serves as a positive regulator of erythroid differentiation.
Other Names:
T-cell acute lymphocytic leukemia protein 1, TAL-1, Class A basic helix-loop-helix protein 17, bHLHa17, Stem cell protein, T-cell leukemia/lymphoma protein 5, TAL1, BHLHA17, SCL, TCL5
Target/Specificity:
This TAL1 Antibody is generated from rabbits immunized with a KLH conjugated synthetic phosphopeptide corresponding to amino acid residues surrounding T90 of human TAL1.
Antigen Type: Synthetic Peptide
Gene Name: TAL1
Gene ID: 6886
Primary Accession: P17542
NCBI Accession: NP_003180.1
Format: Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification.
TAL1 is implicated in the genesis of hemopoietic malignancies. It may play an important role in hemopoietic differentiation and serves as a positive regulator of erythroid differentiation.
Other Names:
T-cell acute lymphocytic leukemia protein 1, TAL-1, Class A basic helix-loop-helix protein 17, bHLHa17, Stem cell protein, T-cell leukemia/lymphoma protein 5, TAL1, BHLHA17, SCL, TCL5
Target/Specificity:
This TAL1 Antibody is generated from rabbits immunized with a KLH conjugated synthetic phosphopeptide corresponding to amino acid residues surrounding T90 of human TAL1.
Antigen Type: Synthetic Peptide
Gene Name: TAL1
Gene ID: 6886
Primary Accession: P17542
NCBI Accession: NP_003180.1
Format: Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification.
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